pylori in English
A septum of connective tissue separates the circular muscle layers of the pylorus and duodenum.
Use "pylori" in a sentence
1. H pylori infection was a strong predictor of ulcer recurrences.
2. Helicobacter pylori infection is the main causative factor in stomach cancer.
3. H pylori was identified by culture and histology as previously described.
4. H. pylori lives only in the slimy, acidic mucus inside our stomachs.
5. H. pylori infection speeds up that cancer progression , to about seven months .
6. Eradication of H. pylori is an effective optional treatment for functional dyspepsia.
7. Results Eighty three patients with H pylori positive gastric ulcers entered the study.
8. Active is loosely equivalent to “acute” and often indicates infection by Helicobacter pylori
9. Increased basal serum gastrin is related to atrophy and to infection with H pylori.
10. Aim To reveal the virulence variation related to vacuolating cytotoxicity of coccoid Helicobacter pylori .
11. These findings support the hypothesis that H pylori colonisation is directly involved in the hypergastrinaemia.
12. Pylori) is a very common — and yes, Contagious — type of bacteria that infects the digestive tract
13. The H pylori negative and positive healthy volunteers were similar for both basal and peak acid output.
14. 25 Only a minority of patients infected with H pylori will eventually develop a duodenal ulcer.
15. Seven of the parents were positive for H pylori on urease testing, histology, and on culture.
16. 28 There are few data concerning the role of anti- H pylori treatment in gastric ulcer disease.
17. The dynamics and importance of H pylori reduced altered mucosal peptic activity is far from being clarified.
18. It is based upon the ability of H. pylori to convert urea to ammonia and carbon dioxide.
19. After eradication of H pylori, duodenal ulcers do not usually recur and the associated chronic gastritis gradually disappears.Sentencedict.com
20. Atrophy, which is closely related to H pylori infection, is associated with a decline in acid secretion.
21. 20 Bismuth has also become popular in recent years as a treatment in peptic ulcer to eradicate Helicobacter pylori.
22. Omeprazole monotherapy merely suppressed bacterial colonisation, especially in the antral region, and eradicated H pylori in individual cases only.
23. In addition, H pylori eradication speeds up ulcer healing and is associated with healing of previously refractory ulcers.
24. Patients classified as having H pylori infection received an eradication regimen; 107 were reassessed 4 weeks after therapy.
25. 14 The high rate of H pylori ammonia production in uraemic patients should accentuate any ammonia induced effects.
26. Values were too low to judge whether there was any effect associated with Helicobacter pylori colonisation in controls.
27. 21 One of the H pylori negative patients was on maintenance haemodialysis and eight were on continuous ambulatory peritoneal dialysis.
28. In healthy men without atrophy, gastric acid secretion is preserved with ageing and is independent of H pylori status.
29. 13 On logistic regression, the presence and extent of gastric metaplasia was not significantly associated with H pylori infection.
30. These differences persisted when subgroups of patients were analysed according to Helicobacter pylori colonisation or degree of mucosal injury.
31. FeON-FeOFF: the Helicobacter pylori Fur regulator Commutates iron-responsive transcription by discriminative readout of opposed DNA grooves Nucleic Acids Res
32. 17 In conclusion, we investigated the influence of Helicobacter pylori colonisation on gastric mucosal eicosanoid synthesis in patients taking NSAIDs.
33. Of the nine H pylori negative uraemic patients, two had oesophagitis and one had scattered petechiae in stomach and duodenum.
34. 10 On logistic regression,(www.Sentencedict.com) the presence and extent of gastric metaplasia was not significantly associated with H pylori infection.
35. 15 H pylori positive gastritis, and the combination of active duodenitis and gastric metaplasia were independent predictors of duodenal ulceration.
36. Cyclical changes in H pylori infection may cause the variations in basal acid secretion that are seen in duodenal ulcer disease.
37. After eradication of H pylori in the duodenal ulcer patients both their basal acid output and basal gastrin fell by 50%.
38. Abdominal distension also occurs in individuals with lactose intolerance, bacterial food poisoning, parasitic infections like giardia, bacteria like helicobacter pylori (H
39. 16 Thus H pylori infection combined with active ulceration may cause the increased basal acid secretion seen in duodenal ulcer disease.
40. 25 Most investigators have observed that H pylori infection causes a greater percentage increase in the postprandial gastrin than fasting gastrin.
41. 22 Helicobacter pylori is recognised as a significant cause of chronic antral gastritis and important in the aetiology of peptic ulceration.
42. The studies were designed to evaluate the H pylori eradication potency of the various regimens and the post-therapeutic course of ulcer disease.
43. Conclusion: The results revealed significant anti-Helicobacter pylori activity of medicinal plants which could be the potential source of new Bactericidal agents.
44. 22 In conclusion, our findings suggest that H pylori infection may have been predominantly acquired at a young age in the past.
45. 18 The primary aim of the second trial was to see if eradication of H pylori led to resolution of intestinal metaplasia.
46. 8 It has also been proposed that H pylori ammonia production will lead to mucosal damage by denaturing the protective mucus layer.
47. Discussion Our current results confirm that treatment which both heals duodenal ulcers and eradicates H pylori significantly decreases basal plasma gastrin concentrations.
48. 30 The studies were designed to evaluate the H pylori eradication potency of the various regimens and the post-therapeutic course of ulcer disease.
49. And recently, we used to think that stomach ulcers were just caused by stress, until we discovered that H. pylori was the culprit.
50. 16 The role of the increased serum gastrin concentration induced by H pylori in the pathogenesis of duodenal ulcer disease is also unknown.