1. Isoproterenol to increase heart rate can be tried cautiously until pacing is instituted.

2. The acute toxicity of isoproterenol is much less than that of epinephrine.

3. Propranolol reduced heart rate as expected and blocked the responses to isoproterenol administration.

4. Slow action potentials were induced by 10−6 M isoproterenol with electrical stimulation.

5. This accelerating effect of isoproterenol on the regeneration of the Ca-mediated action potential was mimicked by theophylline.

6. These results are consistent with those of other investigators showing a suppressibility of the isoproterenol-induced pancreatic amylase secretion by atropine.

7. The observed isoproterenol-stimulated changes in cyclic-AMP metabolism of the ventral prostate were time-dependent and maximal stimulation was seen 5 min after treatment with this β-adrenergic agonist.

8. These Catecholamines can interact with α and β receptors of the cardiovascular systems (Ramos et al., 1996).Some Catecholamines, such as isoproterenol, have been developed for the treatment of

9. Other Catecholamines occur in trace amounts in the body, and synthetic Catecholamines are available; for example isoprenaline (isoproterenol) was previously employed for the relief of asthma, while α-methylDOPA reduces blood …

10. Forskolin, a direct activator of adenylate cyclase, and isoproterenol, a β-adrenergic agonist, increased intracellular cyclic AMP levels in a concentration-dependent manner, subsequent to a 5-min exposure.

11. Owing to the positive inotropic effect of noradrenaline and isoproterenol the relative increase in contractile force at rising frequencies proved less pronounced than in control experiments, although the absolute maximal force that developed was higher in presence of the sympathomimetic drugs.

12. It has been shown that bromocriptine-induced tachycardia, which persisted after adrenalectomy, is (i) mediated by central dopamine D2 receptor activation and (ii) reduced by 5-day isoproterenol pretreatment, supporting therefore the hypothesis that this effect is dependent on sympathetic outflow to the heart.