duodenal in English

adjective

[du·o·de·nal || ‚djuːəʊ'diːnl]

pertaining to the duodenum (part of the small intestine)

Use "duodenal" in a sentence

1. Conclusions Combined biliary and duodenal stenting is an effective method palliation of biliary and duodenal obstructions.

2. 5 An ulcer perforates the duodenal wall.

3. Stimulation of one or both vagi at 5 or 15 Hz or field stimulation of intrinsic duodenal nerves produced Significant increases in duodenal vein IR motilin concentrations and accompanying increases in duodenal motility.

4. Indications are mainly obstructive jaundice, duodenal obstruction and pain.

5. How is duodenal Atresia diagnosed? Duodenal Atresia is diagnosed by ultrasound, but not usually at the routine 20-week screening ultrasound.

6. In duodenal Atresia, there is only one obstruction

7. 11 The main, unsolved clinical problem is chronic duodenal ulcer.

8. Duodenal Atresia is classified into three types: type I (duodenal diaphragm) is linked to the presence of a mucosal diaphragmatic membrane with an intact muscle wall; type II (complete a duodenal Atresia) characterized by a short fibrous cord connecting the two ends of the atresic duodenum; and type III (also complete duodenal Atresia) which

9. Duodenal Atresia – Duodenal Atresia is when the blockage is located in the duodenum, which is at the beginning of the small intestine near the stomach

10. Cicatrised duodenal ulcer 21 52.5 Carcinoma pyloric antrum 09 22.5 Total 40 100 Cicatrized duodenal ulcer was the most common cause of gastric outlet obstruction

11. Pylori-related gastritis in the Antrum strongly correlates with an increased risk of peptic ulcer disease, and of duodenal ulcer in particular (the duodenal ulcer phenotype of gastritis)

12. Other sites of varices, including esophageal varices, duodenal varices and colonic varices.

13. 18 A bacterium is identified as the cause for his duodenal ulcer.

14. Some babies with duodenal Atresia may also be diagnosed with Down syndrome.

15. Duodenal Atresia is a congenital condition, which means it develops before birth

16. It's a big duodenal hematoma with pretty brisk bleeding along the anterior perforation.

17. The frequency of duodenal adenomas in patients with, familial adenomatous polyposis is high.

18. Symptoms Cicatrised duodenal ulcer Carcinoma antrum Total Number Percentage Number Percentage Number Percentage

19. Heterotopia of the pancreas tissue (Acinuses—A and ducts—D) in the duodenal wall

20. 27 Treatment failure was defined as evidence by endoscopy of a recurrent duodenal ulcer crater.

21. 15 Discussion Duodenal ulcer disease has traditionally been associated with excess secretion of gastric acid.

22. The complications of duodenal ulcer that require surgical management are hemorrhage , perforation, obstruction, and intractability.

23. After eradication of H pylori, duodenal ulcers do not usually recur and the associated chronic gastritis gradually disappears.Sentencedict.com

24. 25 Only a minority of patients infected with H pylori will eventually develop a duodenal ulcer.

25. 27 The complications of duodenal ulcer that require surgical management are hemorrhage , perforation, obstruction, and intractability.

26. We described here two cases of active duodenal ulcers associated with linitis plastica of the antrum.

27. Duodenal and Ampullary masses begin as pre-malignant polyps that over time can develop into a malignancy

28. 13 In conclusion, dietary polyunsaturated fatty acids enhance duodenal resistance to acid by potentiation of adaptive cytoprotection.

29. 13 Eighty unrelated controls, 61 patients with gastric ulcer, and 57 patients with duodenal ulcer were studied.

30. Li died of duodenal cancer in Beijing at 78, in the darkest period of the Cultural Revolution.

31. Gastrin levels may also be elevated by pernicious anemia. duodenal ulcers[sentencedict .com], and after a meal.

32. 9 In conclusion, dietary polyunsaturated fatty acids enhance duodenal resistance to acid by potentiation of adaptive cytoprotection.

33. In cases of delayed diagnosis, we recommend drainage of the perforation, naso-duodenal suction tube, parenteral alimentation.

34. 2 According to previous studies, gastric metaplasia is an almost constant finding in patients with duodenal ulceration.

35. In the acid ulcer type (Johnson II and III), duodenal ulcer and anastomotic ulcer supplementary vagotomy is recommendable.

36. 14 If you have many whorls, few loops, patterned palms the chances are you will develop duodenal ulcers.

37. 28 Objectives Discussing the effect of highly selective vagotomy (HSV) and repair treatment in perforation of duodenal ulcer.

38. Commonest cause of gastric outlet obstruction was carcinoma of pyloric antrum (32) followed by Cicatrised duodenal ulcer (26)

39. The initial vagotomy was done for duodenal ulcer (23), gastric ulcer (17), and acid dyspepsia without ulcer (4).

40. 28 No other model of chronic ulcer shows such morphological and behavioural similarity to the human duodenal ulcer.

41. Cholecystokinin definition: a hormone secreted by duodenal cells that stimulates the contraction of the gall bladder Meaning, pronunciation, translations and examples

42. The Bilio-pancreatic limb plays an important role in the beneficial effect of duodenal-jejunal bypass for nonalcoholic steatohepatitis

43. Commonest cause of gastric outlet obstruction in our study was carcinoma of pyloric antrum followed by Cicatrised duodenal ulcer.

44. 15 H pylori positive gastritis, and the combination of active duodenitis and gastric metaplasia were independent predictors of duodenal ulceration.

45. Gallstone colics occurred more often with gallbladder- and porta hepatis carcinoma than with common bile tract- and duodenal papilla carcinoma.

46. 7 To determine the extent of gastric metaplasia, multiple biopsy specimens were collected from standardised sites of the duodenal bulb.

47. Subsequent accumulation of duodenal alkaline phosphatase activity (EC 3.1.3.1) and protein content were monitored as developmental parameters (n = 8–43).

48. 16 Phosphatidylglycerol was detectable in patients with chronic atrophic gastritis, but not in controls or in patients with duodenal ulcer.

49. 15 As a result, the inflamed duodenal mucosa may then become susceptible to acid and this may ultimately lead to ulceration.

50. After eradication of H pylori in the duodenal ulcer patients both their basal acid output and basal gastrin fell by 50%.